what are the expected signs of compartment syndrome

ATI RN

ATI Capstone Medical Surgical Assessment 1 Quizlet

1. What are the expected signs of compartment syndrome?

A. Unrelieved pain, pallor, and pulselessness
B. Fever, swelling, and redness
C. Muscle cramps and weakness
D. Redness and itching

Correct answer: A

Rationale: The correct answer is A: Unrelieved pain, pallor, and pulselessness. Compartment syndrome is characterized by increased pressure within a muscle compartment, leading to inadequate blood supply. This results in severe pain that is unrelieved by rest or medication, pallor due to compromised blood flow, and pulselessness as a late sign of severe ischemia. Choices B, C, and D are incorrect. Fever, swelling, and redness are not typical signs of compartment syndrome. Muscle cramps and weakness may occur due to other conditions, but they are not primary indicators of compartment syndrome. Redness and itching are also not commonly associated with compartment syndrome.

2. What dietary instructions should be provided for a patient with pre-dialysis end-stage kidney disease?

A. Limit phosphorus intake to 700 mg/day
B. Increase protein intake to 1g/kg/day
C. Increase sodium intake
D. Avoid potassium-rich foods

Correct answer: A

Rationale: Patients with pre-dialysis end-stage kidney disease should limit phosphorus intake to manage their condition. Excessive phosphorus can lead to mineral and bone disorders in patients with kidney disease. Choices B, C, and D are incorrect. Increasing protein intake is not recommended as it can burden the kidneys. Increasing sodium intake is usually discouraged due to its association with hypertension and fluid retention in kidney disease. Avoiding potassium-rich foods is more relevant in advanced kidney disease stages when potassium levels are high, not in pre-dialysis end-stage kidney disease.

3. What is the priority dietary modification for a patient with pre-dialysis end-stage kidney disease?

A. Limit phosphorus intake to 700 mg/day
B. Increase potassium intake to 3 g/day
C. Eat three large meals per day
D. Restrict protein intake to 1 g/kg/day

Correct answer: A

Rationale: The correct answer is A: Limit phosphorus intake to 700 mg/day. In patients with pre-dialysis end-stage kidney disease, restricting phosphorus intake is crucial to manage their condition. Excessive phosphorus can lead to mineral and bone disorders, which are common in kidney disease. Choice B, increasing potassium intake, is not the priority and can be harmful as kidney disease often leads to hyperkalemia. Choice C, eating three large meals per day, is not recommended as smaller, frequent meals are usually better tolerated. Choice D, restricting protein intake to 1 g/kg/day, is important in later stages of kidney disease but is not the priority at the pre-dialysis stage.

4. What is a characteristic sign of hypokalemia on an ECG?

A. Flattened T waves
B. ST elevation
C. Prominent U waves
D. Widened QRS complex

Correct answer: A

Rationale: Flattened T waves are a characteristic sign of hypokalemia on an ECG. When potassium levels are low, it can lead to changes in the ECG, such as T wave flattening. This alteration is important to recognize as it indicates potential electrolyte imbalances. ST elevation (Choice B) is not typically associated with hypokalemia but can be seen in conditions like myocardial infarction. Prominent U waves (Choice C) are associated with hypokalemia, but flattened T waves are more specific. Widened QRS complex (Choice D) is not a typical ECG finding in hypokalemia but can be seen in conditions like hyperkalemia.

5. What ECG changes are expected in hypokalemia?

A. Flattened T waves on ECG
B. Prominent U waves on ECG
C. Widened QRS complexes on ECG
D. ST elevation on ECG

Correct answer: A

Rationale: In hypokalemia, flattened T waves are a common ECG finding due to the decreased extracellular potassium affecting repolarization. Prominent U waves are typically seen in hypokalemia as well, but flattened T waves are the more specific and early ECG change. Widened QRS complexes are associated with hyperkalemia, not hypokalemia. ST elevation is often seen in conditions like myocardial infarction, pericarditis, or early repolarization syndrome, not specifically in hypokalemia.