what causes secondary brain injury after head trauma

ATI RN

ATI Pathophysiology Exam 1

1. What causes secondary brain injury after head trauma?

A. Brain injury resulting from the body’s response to tissue damage
B. Brain injury resulting from initial trauma
C. Injury as a result of medical therapy
D. Focal areas of bleeding

Correct answer: A

Rationale: The correct answer is A. Secondary brain injury occurs due to the body's response to the initial trauma, which can worsen the effects of the primary injury. This response includes processes like inflammation, increased intracranial pressure, and reduced oxygen delivery to tissues. Choice B is incorrect because it refers to the primary trauma itself, not the secondary injury. Choice C is incorrect as it relates to injury caused by medical interventions rather than the body's response. Choice D is incorrect as it specifically mentions focal areas of bleeding, which is a consequence of trauma rather than the cause of secondary brain injury.

2. Which of the following is a characteristic of osteosarcoma?

A. Slow-growing tumor that begins in the bone marrow
B. Solitary tumor that most often affects the metaphyseal region of the femur or tibia
C. An aggressive tumor most often found in the bone marrow of long bones
D. A tumor that infiltrates the trabeculae in spongy bone and implants in surrounding tissue

Correct answer: C

Rationale: Osteosarcoma is an aggressive bone tumor that most commonly arises in the bone marrow of long bones, such as the femur or tibia. Choice A is incorrect as osteosarcoma is not a slow-growing tumor but rather a fast-growing one. Choice B is incorrect as osteosarcoma is not typically solitary but can involve multiple lesions. Choice D is incorrect as osteosarcoma primarily affects the medullary (marrow) cavity of bones, rather than infiltrating the trabeculae in spongy bone and surrounding tissue.

3. A male patient is receiving androgen therapy for hypogonadism. What adverse effect should the nurse monitor for during this therapy?

A. Increased risk of bone fractures
B. Increased risk of cardiovascular events
C. Increased risk of liver dysfunction
D. Increased risk of prostate cancer

Correct answer: B

Rationale: The correct answer is B: Increased risk of cardiovascular events. Androgen therapy can lead to an increased risk of cardiovascular events like heart attacks and strokes, especially in older patients. Monitoring for signs and symptoms of cardiovascular issues is essential during this therapy. Choices A, C, and D are incorrect because androgen therapy is not typically associated with an increased risk of bone fractures, liver dysfunction, or prostate cancer.

4. In which of the following cases is dehydration more likely to occur?

A. A 50-year-old woman who is a bodybuilder
B. A 50-year-old man who is morbidly obese
C. A 10-year-old child who is thin
D. A 30-year-old with multiple sclerosis

Correct answer: B

Rationale: The correct answer is B. Morbid obesity increases the risk of dehydration due to the larger body surface area and potential for greater insensible losses. Choices A, C, and D are less likely to experience dehydration compared to a morbidly obese individual.

5. What function does aldosterone serve in the body?

A. Aldosterone causes a release of sodium from the body, decreases fluid volume, and decreases blood pressure
B. Aldosterone causes a retention of sodium in the body, increases fluid volume, and increases blood pressure
C. Aldosterone causes a release of sodium from the body, increases fluid volume, and decreases blood pressure
D. Aldosterone enhances intracellular sodium production and lowers blood pressure

Correct answer: B

Rationale: Aldosterone functions by causing the retention of sodium in the body, which results in an increase in fluid volume and blood pressure. Choice A is incorrect because aldosterone actually promotes sodium retention rather than release. Choice C is incorrect as it states that aldosterone decreases fluid volume, which is not accurate. Choice D is incorrect because aldosterone does not enhance intracellular sodium production; instead, it primarily acts on sodium reabsorption in the kidneys.