what are the ecg changes seen with hyperkalemia

ATI RN

ATI Capstone Adult Medical Surgical Assessment 2

1. What ECG changes are seen with hyperkalemia?

A. Flattened T waves
B. Elevated ST segments
C. Prominent U waves
D. Widened QRS complex

Correct answer: A

Rationale: Flattened T waves are an early ECG sign of hyperkalemia. Hyperkalemia affects the repolarization phase of the cardiac action potential, leading to changes such as peaked T waves, prolonged PR interval, widened QRS complex, and ultimately sine wave pattern. Elevated ST segments, prominent U waves, and widened QRS complex are not typically associated with hyperkalemia, making choices B, C, and D incorrect.

2. What are the expected signs in a patient with compartment syndrome?

A. Unrelieved pain, pallor, and pulselessness
B. Fever and infection
C. Localized redness and swelling
D. Loss of sensation in the affected area

Correct answer: A

Rationale: In a patient with compartment syndrome, the classic signs include unrelieved pain, pallor, and pulselessness. These signs indicate compromised circulation and neurovascular function in the affected compartment. Fever and infection (Choice B) are not typical signs of compartment syndrome. Localized redness and swelling (Choice C) can be seen in other conditions like cellulitis but are not specific to compartment syndrome. Loss of sensation in the affected area (Choice D) may occur in some cases but is not as specific or consistent as unrelieved pain, pallor, and pulselessness in diagnosing compartment syndrome.

3. What is the preferred electrical intervention for a patient with ventricular tachycardia and a pulse?

A. Synchronized cardioversion
B. Defibrillation
C. Medication administration
D. Pacing

Correct answer: A

Rationale: In a patient with ventricular tachycardia and a pulse, synchronized cardioversion is the preferred electrical intervention. Synchronized cardioversion is used to treat unstable tachyarrhythmias like ventricular tachycardia with a pulse. Choice B, defibrillation, is used for pulseless ventricular tachycardia or ventricular fibrillation. Choice C, medication administration, may not provide immediate correction for unstable ventricular tachycardia. Choice D, pacing, is not the first-line treatment for ventricular tachycardia with a pulse.

4. What are the expected manifestations of a thrombotic stroke?

A. Gradual loss of function on one side of the body
B. Loss of sensation in the affected extremity
C. Sudden loss of consciousness
D. Seizures and convulsions

Correct answer: A

Rationale: The correct answer is A: Gradual loss of function on one side of the body. Thrombotic strokes are caused by a clot forming in a blood vessel supplying the brain, leading to a gradual onset of symptoms due to impaired blood flow to specific brain regions. Choices B, C, and D are incorrect because loss of sensation, sudden loss of consciousness, seizures, and convulsions are not typically associated with thrombotic strokes. In a thrombotic stroke, the symptoms develop slowly over time, often over minutes to hours, and include manifestations such as weakness, numbness, or paralysis on one side of the body, along with other symptoms related to the affected brain area.

5. What are the expected manifestations in a patient experiencing a thrombotic stroke?

A. Sudden numbness or loss of function on one side of the body
B. Sudden loss of consciousness and seizure
C. Gradual onset of difficulty speaking
D. Loss of sensation in the affected limb

Correct answer: A

Rationale: The correct manifestation in a patient experiencing a thrombotic stroke is sudden numbness or loss of function on one side of the body. This is due to the blockage of a blood vessel by a clot, leading to a lack of blood flow to a specific part of the brain. Choices B, C, and D are incorrect. Sudden loss of consciousness and seizure are more commonly associated with hemorrhagic strokes. Gradual onset of difficulty speaking is often seen in ischemic strokes affecting language areas, not specifically in thrombotic strokes. Loss of sensation in the affected limb is more indicative of sensory nerve damage rather than the motor deficits seen in thrombotic strokes.