Nursing Elites

1. The client has recently been diagnosed with irritable bowel syndrome (IBS). Which intervention should the nurse teach the client to reduce symptoms?

• A. Instruct the client to avoid drinking fluids with meals
• B. Explain the need to decrease intake of flatus-forming foods
• C. Teach the client how to perform gentle perianal care
• D. Encourage the client to see a psychologist

Correct answer: B

Rationale: The correct answer is B. Decreasing the intake of flatus-forming foods can help reduce symptoms of bloating and discomfort in IBS. This intervention focuses on dietary modifications that can positively impact the client's condition. Instructing the client to avoid drinking fluids with meals (choice A) may not directly address the underlying cause of IBS symptoms. Teaching perianal care (choice C) is important for hygiene but does not directly address IBS symptoms. Encouraging the client to see a psychologist (choice D) may be beneficial for managing stress or anxiety associated with IBS but does not directly target symptom reduction through dietary changes.

2. The nurse understands that which are characteristics of anthrax? Select all that apply.

• A. Cutaneous lesions become a black eschar, Flu-like symptoms are a sign of pulmonary anthrax
• B. Cutaneous lesions become a black eschar
• C. Gastrointestinal anthrax causes blood anthrax
• D. Flu-like symptoms are a sign of pulmonary anthrax

Correct answer: A

Rationale: The correct characteristics of anthrax are that cutaneous lesions become a black eschar, and flu-like symptoms are typical of pulmonary anthrax. Choice B is incorrect as it only covers the cutaneous anthrax characteristic and does not include the flu-like symptoms of pulmonary anthrax. Choice C is incorrect as gastrointestinal anthrax does not cause 'blood anthrax,' and Choice D is incorrect as flu-like symptoms are not associated with gastrointestinal anthrax.

3. The two members of the health care team who work closely to monitor drug-nutrient interactions are:

• A. Physician and nurse
• B. Physician and pharmacist
• C. Nurse and clinical dietitian
• D. Clinical dietitian and pharmacist

Correct answer: D

Rationale: Clinical dietitians and pharmacists are the key members of the healthcare team responsible for monitoring drug-nutrient interactions. Clinical dietitians assess patients' nutritional needs and develop appropriate diets that consider medication effects, while pharmacists provide expertise on medications and their interactions with nutrients. Physicians and nurses are essential healthcare providers but typically do not have the specialized knowledge required to manage drug-nutrient interactions, making choices A, B, and C incorrect.

4. One of the reasons hospital patients are at greater risk for drug-nutrient interactions than they used to be is because:

• A. Hospitalized patients are more acutely ill
• B. Hospital routines interfere with the correct timing of medications
• C. Drugs used today are more toxic and have more side effects
• D. Responsibility for monitoring this is shared by various members of the healthcare team

Correct answer: A

Rationale: The correct answer is A. Hospitalized patients are more acutely ill, often having multiple conditions and treatments, which increases the risk of drug-nutrient interactions. Choice B is incorrect because hospital routines do not specifically interfere with the timing of medications in relation to drug-nutrient interactions. Choice C is incorrect because the toxicity and side effects of drugs do not directly relate to an increased risk of drug-nutrient interactions. Choice D is incorrect as sharing responsibility for monitoring does not inherently increase the risk of drug-nutrient interactions in hospital patients.

5. What causes hepatic encephalopathy?

• A. Buildup of ammonia in the body
• B. Buildup of urea in the body
• C. Fatty infiltration of the liver
• D. Jaundice

Correct answer: A

Rationale: Hepatic encephalopathy is caused by the buildup of ammonia in the body. Ammonia, a byproduct of protein metabolism, normally gets converted to urea in the liver for excretion. However, in liver dysfunction, such as cirrhosis, the liver cannot effectively convert ammonia to urea, leading to its accumulation in the body and subsequently causing hepatic encephalopathy. Choices B, C, and D are incorrect as they do not directly relate to the pathophysiology of hepatic encephalopathy.

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