Nursing Elites

1. In assessing the client's chest, which position best shows chest expansion as well as its movements?

• A. Sitting
• B. Prone
• C. Sidelying
• D. Supine

Correct answer: A

Rationale: The correct answer is A: Sitting. When the client is seated, their chest is in an optimal position for observing the full range of chest expansion during breathing. This position allows for easy visualization of chest movements and expansion as the client breathes in and out, providing a comprehensive assessment of respiratory function. Choice B, Prone, is incorrect as lying face down would not provide a clear view of chest expansion. Choice C, Sidelying, is also incorrect as this position may limit the visibility of chest movements. Choice D, Supine, is not the best position for assessing chest expansion as it may not offer a clear observation of chest movements during breathing.

2. Which laboratory data indicate the client’s pancreatitis is improving?

• A. The amylase and lipase serum levels are decreased
• B. The white blood cell count (WBC) is decreased
• C. The conjugated and unconjugated bilirubin levels are decreased
• D. The blood urea nitrogen (BUN) serum level is decreased

Correct answer: A

Rationale: The correct answer is A. Amylase and lipase are specific markers for pancreatitis. A decrease in their serum levels indicates improvement in pancreatitis. Choice B, a decreased white blood cell count (WBC), is more indicative of an improvement in infection rather than pancreatitis. Choices C and D, decreased bilirubin levels and blood urea nitrogen (BUN) levels respectively, are not specific markers for pancreatitis improvement.

3. Students in the resident M6 Practical Nurse Course are expected to achieve entry-level competencies for which of the following?

• A. Medical-surgical nursing
• B. Obstetric and newborn nursing
• C. Pediatric nursing
• D. Trauma nursing

Correct answer: A

Rationale: The correct answer is A: Medical-surgical nursing. In the resident M6 Practical Nurse Course, students are expected to achieve entry-level competencies in medical-surgical nursing. This area of nursing focuses on caring for adult patients with a variety of medical conditions. Obstetric and newborn nursing (choice B), pediatric nursing (choice C), and trauma nursing (choice D) are specialized areas within nursing that are not typically covered in entry-level practical nurse courses, making them incorrect choices.

4. Why are hospital patients at greater risk for drug-nutrient interactions than they used to be?

• A. Hospitalized patients are more acutely ill
• B. Hospital routines interfere with the correct timing of medications
• C. Drugs used today are more toxic and have more side effects
• D. Responsibility for monitoring this is shared by various members of the healthcare team

Correct answer: A

Rationale: The correct answer is A. Hospitalized patients are at greater risk for drug-nutrient interactions because they are more acutely ill, often having multiple conditions and treatments that increase the risk of such interactions. Choice B is incorrect as hospital routines interfering with medication timing are not directly related to drug-nutrient interactions. Choice C is incorrect as the toxicity and side effects of drugs do not necessarily relate to interactions with nutrients. Choice D is incorrect as shared responsibility for monitoring does not directly contribute to the increased risk of drug-nutrient interactions in hospitalized patients.

5. What causes hepatic encephalopathy?

• A. Buildup of ammonia in the body
• B. Buildup of urea in the body
• C. Fatty infiltration of the liver
• D. Jaundice

Correct answer: A

Rationale: Hepatic encephalopathy is caused by the buildup of ammonia in the body. Ammonia, a byproduct of protein metabolism, normally gets converted to urea in the liver for excretion. However, in liver dysfunction, such as cirrhosis, the liver cannot effectively convert ammonia to urea, leading to its accumulation in the body and subsequently causing hepatic encephalopathy. Choices B, C, and D are incorrect as they do not directly relate to the pathophysiology of hepatic encephalopathy.

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