why must a child with acute laryngotracheobronchitis be kept npo

HESI LPN

Nutrition Final Exam Quizlet

1. Why must a child with acute laryngotracheobronchitis be kept NPO?

A. The epinephrine administration may cause nausea and vomiting
B. The child is being hydrated with IV fluids
C. The child may not feel hungry
D. Rapid respirations pose a risk for aspiration

Correct answer: D

Rationale: In acute laryngotracheobronchitis, rapid respirations increase the risk of aspiration due to compromised airway protection and potential for secretions to enter the lungs. Keeping the child NPO helps prevent the risk of aspiration pneumonia. Choice A is incorrect because epinephrine is not typically used for laryngotracheobronchitis. Choice B is incorrect as hydration with IV fluids does not eliminate the risk of aspiration. Choice C is also incorrect because the child being hungry is not the primary reason for keeping them NPO in this condition.

2. What is an important aspect of care for a child with congenital adrenal hyperplasia?

A. Monitoring electrolyte levels
B. Restricting physical activity
C. Administering high doses of vitamin A
D. Providing a high-protein diet

Correct answer: A

Rationale: The correct answer is A: Monitoring electrolyte levels. In the management of congenital adrenal hyperplasia, it is crucial to monitor electrolyte levels to prevent imbalances and related complications. Restricting physical activity (choice B) is not a typical aspect of care for this condition. Administering high doses of vitamin A (choice C) is not a standard treatment for congenital adrenal hyperplasia and can be harmful in excess. Providing a high-protein diet (choice D) may be beneficial for some conditions but is not a specific requirement for managing congenital adrenal hyperplasia.

3. What is a common symptom of a vitamin D deficiency in children?

A. Frequent infections
B. Bone pain and tenderness
C. Excessive thirst
D. Unexplained bruising

Correct answer: B

Rationale: Bone pain and tenderness are indeed common symptoms of vitamin D deficiency in children. Vitamin D plays a crucial role in bone health and growth. The deficiency can lead to weakened bones, causing pain and tenderness. Choices A, C, and D are incorrect. Frequent infections are not a typical symptom of vitamin D deficiency in children; excessive thirst is more commonly associated with conditions like diabetes, and unexplained bruising is not directly linked to vitamin D deficiency.

4. What is the recommended treatment for a child with a first-degree burn?

A. Immediate application of ice
B. Administration of oral antibiotics
C. Application of cool, moist compresses
D. Application of hot packs

Correct answer: C

Rationale: The recommended treatment for a child with a first-degree burn is the application of cool, moist compresses. This helps in reducing pain and swelling without causing further damage to the skin. Applying ice directly to a burn can lead to frostbite and worsen the injury (Choice A). Oral antibiotics are not necessary for first-degree burns as they do not typically involve infection (Choice B). Hot packs should be avoided as they can further damage the skin and increase inflammation (Choice D).

5. What dietary recommendation is essential for a child with phenylketonuria (PKU)?

A. High-protein diet
B. Low-fat diet
C. Low-phenylalanine diet
D. High-fiber diet

Correct answer: C

Rationale: The correct answer is C: 'Low-phenylalanine diet.' Children with phenylketonuria (PKU) need to follow a low-phenylalanine diet to manage the condition. Phenylalanine is an amino acid found in protein-containing foods, and individuals with PKU have difficulty metabolizing it, leading to neurological damage and other complications. Therefore, restricting phenylalanine intake is crucial. Choice A, 'High-protein diet,' is incorrect because high protein intake would increase phenylalanine levels, worsening the condition. Choice B, 'Low-fat diet,' and Choice D, 'High-fiber diet,' are not the primary focus for PKU patients. The key dietary intervention for PKU is controlling phenylalanine intake, which is best achieved through a low-phenylalanine diet.