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HESI Pediatrics Quizlet
1. What factor predisposes the urinary tract to infection in children?
- A. increased fluid intake
- B. short urethra in young girls
- C. prostatic secretions in males
- D. frequent emptying of the bladder
Correct answer: B
Rationale: The short urethra in young girls predisposes them to urinary tract infections. In young girls, the proximity of the urethra to the anus and the shorter urethra compared to boys make it easier for bacteria to travel up the urinary tract, increasing the risk of infection. Increased fluid intake and frequent emptying of the bladder are actually helpful in preventing urinary tract infections by flushing out bacteria. Prostatic secretions in males are not a factor in predisposing the urinary tract to infection in children.
2. When obtaining a health history from parents of a 4-month-old boy with congenital hypothyroidism, what would the nurse most likely assess?
- A. The child's growth is above normal for his age.
- B. The child is active and playful.
- C. The skin appears pink and healthy.
- D. It is difficult to keep the child awake.
Correct answer: D
Rationale: In congenital hypothyroidism, infants often experience lethargy and difficulty staying awake due to low thyroid hormone levels. Choice A is incorrect as hypothyroidism can lead to poor growth in infants. Choice B is incorrect because hypothyroidism can cause decreased activity levels and lethargy rather than being active and playful. Choice C is incorrect as hypothyroidism can result in dry skin and poor skin tone, not necessarily pink and healthy-looking skin.
3. Why does a cleft lip predispose an infant to infection, concerning a nurse caring for the infant?
- A. Waste products accumulate along the defect.
- B. There is inadequate circulation in the defective area.
- C. Nutrition is inadequate due to ineffective feeding.
- D. Mouth breathing dries the oropharyngeal mucous membranes.
Correct answer: D
Rationale: Mouth breathing due to a cleft lip can dry the mucous membranes, increasing their susceptibility to infection. While waste product accumulation (Choice A) and inadequate circulation (Choice B) may contribute to complications, they are not directly related to infection in this context. Inadequate nutrition (Choice C) may affect overall health but is not the primary reason for infection predisposition in this case.
4. The nurse notes that a child has lost 8 pounds after 4 days of hospitalization for acute glomerulonephritis. This is most likely the result of
- A. poor appetite
- B. increased potassium intake
- C. reduction of edema
- D. restriction to bed rest
Correct answer: C
Rationale: The correct answer is C: reduction of edema. In acute glomerulonephritis, weight loss is often a result of the reduction of edema. Acute glomerulonephritis causes fluid retention and edema due to kidney inflammation. As the inflammation resolves with treatment, the kidneys can excrete excess fluid, leading to weight loss. Choices A, B, and D are incorrect. Poor appetite, increased potassium intake, and restriction to bed rest are not typically the primary reasons for weight loss in acute glomerulonephritis.
5. Which is the most appropriate nursing diagnosis for the child with acute glomerulonephritis?
- A. Risk for injury related to malignant process and treatment
- B. Fluid volume deficit related to excessive losses
- C. Fluid volume excess related to decreased plasma filtration
- D. Fluid volume excess related to fluid accumulation in tissues and third spaces
Correct answer: C
Rationale: The most appropriate nursing diagnosis for a child with acute glomerulonephritis is fluid volume excess related to decreased plasma filtration. Acute glomerulonephritis is characterized by inflammation of the glomeruli in the kidneys, leading to decreased plasma filtration and retention of fluid. This results in fluid volume excess rather than fluid deficit (choice B) or fluid accumulation in tissues and third spaces (choice D). The diagnosis of 'risk for injury related to malignant process and treatment' (choice A) is not directly related to the pathophysiology of acute glomerulonephritis.
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