Nursing Elites

1. How is resistant starch digested in the colon?

• A. bacterial fermentation.
• B. pancreatic amylase.
• C. hydrochloric acid.
• D. villi and microvilli.

Correct answer: A

Rationale: In the colon, resistant starch is digested by bacterial fermentation. The correct answer is A. During this process, short-chain fatty acids are produced. Pancreatic amylase, as mentioned in choice B, is responsible for breaking down starch in the small intestine, not in the colon. Choice C, hydrochloric acid, functions in the stomach to aid in the digestion of proteins, not starch. Villi and microvilli, as stated in choice D, are structures in the small intestine that absorb nutrients; they do not participate in the digestion of resistant starch in the colon.

2. The interrelated network of nerves within the gastrointestinal wall that regulates its muscular action is known as the:

• A. gastric nerve plexus.
• B. biliary nerve plexus.
• C. intramural nerve plexus.
• D. intestinal nerve plexus.

Correct answer: C

Rationale: The correct answer is C - intramural nerve plexus. This network of nerves regulates the muscular movements within the gastrointestinal wall. The gastric nerve plexus (choice A) specifically relates to the stomach, not the entire gastrointestinal tract. The biliary nerve plexus (choice B) is associated with the bile ducts and gallbladder, not the regulation of muscular action. The intestinal nerve plexus (choice D) is a general term that doesn't specifically refer to the network of nerves within the gastrointestinal wall.

3. The type of protein-energy malnutrition that results in edema, hypoalbuminemia, skin lesions, and fatty liver is:

• A. cachexia.
• B. marasmus.
• C. kwashiorkor.
• D. sarcopenia.

Correct answer: C

Rationale: The correct answer is C, kwashiorkor. Kwashiorkor is a type of protein-energy malnutrition characterized by edema, hypoalbuminemia, skin lesions, and fatty liver. Edema is a key feature of kwashiorkor due to hypoalbuminemia, which leads to decreased oncotic pressure. Marasmus (choice B) is a form of severe malnutrition characterized by energy deficiency without significant protein deficiency, resulting in severe wasting. Cachexia (choice A) is a syndrome characterized by weight loss, muscle atrophy, fatigue, weakness, and loss of appetite in someone who is not actively trying to lose weight. Sarcopenia (choice D) is the age-related loss of muscle mass and function.

4. In addition to protein, which nutrients contribute to the building and repair of tissue?

• A. Vitamins
• B. Carbohydrates
• C. Fats
• D. Enzymes

Correct answer: A

Rationale: The correct answer is A: Vitamins. Vitamins play a crucial role in tissue building and repair along with protein. Carbohydrates (choice B) are primarily a source of energy and not directly involved in tissue building. Fats (choice C) are important for energy storage and insulation but are not the main contributors to tissue building. Enzymes (choice D) are biological catalysts and do not directly contribute to tissue building and repair.

5. The type of protein-energy malnutrition (PEM) that develops when the diet lacks protein is called:

• A. kwashiorkor.
• B. marasmus.
• C. anemia.
• D. beriberi.

Correct answer: A

Rationale: The correct answer is A: kwashiorkor. Kwashiorkor is a form of protein-energy malnutrition that arises due to a protein-deficient diet. This condition leads to symptoms such as edema, skin lesions, and fatty liver. Choice B, marasmus, is a form of PEM caused by overall malnutrition (protein and calorie deficiency), resulting in severe wasting and muscle loss. Choice C, anemia, is a condition characterized by a deficiency of red blood cells or hemoglobin, not specifically related to protein deficiency. Choice D, beriberi, is a disease caused by thiamine (Vitamin B1) deficiency, not a form of PEM related to protein deficiency.

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