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ATI Nutrition Proctored Exam 2019 Answers
1. Proteins are absorbed primarily in the form of:
- A. fatty acids.
- B. disaccharides.
- C. amino acids.
- D. polypeptides.
Correct answer: C
Rationale: The correct answer is C: amino acids. Proteins are broken down by digestive enzymes into amino acids before being absorbed in the small intestine. Amino acids are the building blocks of proteins and are the form in which they are primarily absorbed. Choice A (fatty acids) is incorrect as fatty acids are the end products of fat digestion, not protein digestion. Choice B (disaccharides) is incorrect because disaccharides are sugars that are broken down into monosaccharides, not proteins. Choice D (polypeptides) is incorrect as proteins are broken down into amino acids and not absorbed as polypeptides.
2. What substance acts as an emulsifier and helps absorb digested fat?
- A. bile
- B. trypsin
- C. lipase
- D. cholecystokinin (CCK)
Correct answer: A
Rationale: The correct answer is A: bile. Bile acts as an emulsifier, breaking down fats into smaller droplets to aid in digestion and absorption. Trypsin is a digestive enzyme that acts on proteins, not fats. Lipase is an enzyme that breaks down fats, but it doesn't act as an emulsifier. Cholecystokinin (CCK) is a hormone that stimulates the release of digestive enzymes, including lipase, but it is not directly involved in emulsification.
3. In which health problem is protein catabolism increased?
- A. severe burns
- B. severe obesity
- C. hypothyroidism
- D. food allergies
Correct answer: A
Rationale: Protein catabolism increases in conditions like severe burns because the body breaks down muscle protein to provide energy for healing. Severe obesity does not necessarily increase protein catabolism; instead, it is more related to excessive fat accumulation. Hypothyroidism may lead to a decrease in metabolic rate but does not directly increase protein catabolism. Food allergies trigger an immune response to specific foods but do not directly impact protein catabolism.
4. Which hormone stimulates the gallbladder to contract?
- A. Secretin.
- B. Cholecystokinin (CCK).
- C. Gastrin.
- D. Gastric inhibitory polypeptide (GIP).
Correct answer: B
Rationale: Cholecystokinin (CCK) is the hormone responsible for stimulating the gallbladder to contract and release bile into the small intestine. Secretin primarily acts on the pancreas to stimulate the secretion of bicarbonate-rich fluid. Gastrin functions in the stimulation of gastric acid secretion, while Gastric inhibitory polypeptide (GIP) plays a role in inhibiting gastric acid secretion and stimulating insulin release.
5. Pepsinogen secreted by the gastric cells is converted into pepsin by:
- A. enterokinase
- B. hydrochloric acid
- C. gastric lipase
- D. pancreatic lipase
Correct answer: B
Rationale: Pepsinogen is converted into its active form, pepsin, by hydrochloric acid in the stomach. Hydrochloric acid helps in unfolding the pepsinogen molecule to transform it into pepsin, which is crucial for protein digestion. Therefore, the correct answer is hydrochloric acid (choice B). Enterokinase (choice A) is an enzyme produced in the small intestine that activates trypsinogen into trypsin, not pepsinogen. Gastric lipase (choice C) is an enzyme that digests fats, not involved in converting pepsinogen to pepsin. Pancreatic lipase (choice D) is an enzyme produced by the pancreas that aids in breaking down fats in the small intestine, not part of the process of pepsin activation.
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